The antiapoptotic protein Api5 and its partner, high molecular weight FGF2, are up-regulated in B cell chronic lymphoid leukemia.

• Authors: KREJČÍ Pavel; PEJCHALOVÁ Kateřina; ROSENBLOOM B.E.; ROSENFELT F.P.; TRAN E.L.; LAURELL H.; WILCOX W.R.
• Year of publication: 2007
• Type: Article in Periodical
• Magazine / Source: Journal of Leukocyte Biology
• MU Faculty or unit: Faculty of Science
• Citation: KREJČÍ, Pavel, Kateřina PEJCHALOVÁ, B.E. ROSENBLOOM, F.P. ROSENFELT, E.L. TRAN, H. LAURELL and W.R. WILCOX. The antiapoptotic protein Api5 and its partner, high molecular weight FGF2, are up-regulated in B cell chronic lymphoid leukemia. Journal of Leukocyte Biology. New York: Society for Leukocyte Biology, 2007, vol. 82, No 6, p. 1363-1364. ISSN 0741-5400. Available from: https://dx.doi.org/10.1189/jlb.0607425.
• Doi: http://dx.doi.org/10.1189/jlb.0607425
• Field: Physiology
• Keywords: FIBROBLAST-GROWTH-FACTOR; api5
• Description: The antiapoptotic protein Api5 and its partner, high molecular weight FGF2, were described to be up-regulated in B cell chronic lymphoid leukemia

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